    When two more characteristics of voice to skull are factored in:

    1. The carrier signal can be "hopped" continuously within the
       bioeffective bandwidth, known as "spread spectrum" transmission,
       and,

    2. The voice modulation most effective for undetectable hypnosis
       is evidently a voice shifted just above normal hearing, but still
       audible to the brain,

    ...you have a recipe for incredibly difficult signals to detect.

    This book is a mainstream publication, very well suited to use
    in our publicity and persuasion campaigns, and our dealings with
    authorities who claim radio signals don't affect living tissue
    except to heat it.

    Finally, many thanks to Blanche Chavoustie for providing me
    photocopies of this book - a saintly work!
    .............................................................
    Page 110:
    ...
    At that time [1953] excitatory mechanisms in nerver fibers
    and nerve cells were grouped under a common rubric of ionic
    equilibrium mechanisms.  There was little interest in the
    possibility that functional organization of mebranes of cell
    bodies might involve threshhold sensitivities to both
    oscillating EM fields and to molecular stimuli at energy
    levels substantially lower than predicted by Hodgkin-
    Huxley models, and substantially below typical threshholds
    in nerve fibers.

    Much recent research cited below has shown that imposed weak
    low frequency fields (and radiofrequency fields amplitude-
    modulated at ELF frequencies) that are many orders of
    magnitude weaker in the pericellular fluid [fluid between
    adjacent cells] than the membrane potential gradient [voltage
    across the membrane] can modulate actions of hormone, anti-
    body neurotransmitter and cancer-promoter molecules at their
    cell surface receptor sites.

    From their electrical characteristics, these sensitivities
    appear to involve nonequilibrium and highly cooperative
    processes that mediate a major amplification of initial
    weak triggers associated with binding of these molecules at
    their specific cell surface receptor sites.

                                    -49-